h3k18ac (MedChemExpress)
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MedChemExpress
h3k18ac
H3k18ac, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/histone+h3/Histone+H3+(acetyl+K18)+Antibody/pm42469847-93-76-78
Average 94 stars, based on 1 article reviews
H3k18ac, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/histone+h3/Histone+H3+(acetyl+K18)+Antibody/pm42469847-93-76-78
Average 94 stars, based on 1 article reviews
h3k18ac - by Bioz Stars,
2026-09
94/100 stars
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Incubation:Article Title: Chlorfenapyr Induces Hepatotoxicity Associated With Oxidative Stress and Mitochondrial Dysfunction Involving Keap1/Nrf2 Signaling. Article Snippet: Equal amounts of protein samples were separated by SDS- PAGE and transferred onto PVDF membranes (ab133411, Abcam). .. Membranes were blocked with 5% skim milk for 1 h at room temperature and then incubated overnight at 4°C with the following primary antibodies: TOM20 (1:5000, A19403, Abclonal), COX IV (1:5000, 11242- 1- AP, Proteintech), CC3 (1:1000, 9661, Cell Signaling Technology), Bax (1:1000, 2772, Cell Signaling Technology), Bcl- 2 (1:1000, PA5- 27094, Thermo Fisher Scientific), Keap1 (1:1000, 4678, Cell Signaling Technology), Nrf2 (1:1000, A21176, Abclonal), HO- 1 (1:1000, PA5- 77833, Thermo Fisher Scientific), NQO1 (1:20,000, A22290, Abclonal), GAPDH (1:10,000, ab181602, Abcam), and Article Title: Topoisomerase I Inhibition in ETV4-overexpressed Non-Small Cell Lung Cancer Promotes Replication and Transcription Mediated R-Loop Accumulation and DNA Damage. Article Snippet: .. In addition, Article Title: Topoisomerase I Inhibition in ETV4‐overexpressed Non‐Small Cell Lung Cancer Promotes Replication and Transcription Mediated R‐Loop Accumulation and DNA Damage Article Snippet: .. In addition, other:Article Title: Caveolin-2 Knockdown Alleviated Sepsis-induced Acute Lung Injury Via Promoting Macrophage M2 Polarization and Inhibiting Apoptosis by Hippo Signaling Pathway Article Snippet: Antibodies against Bax (14796S, CST, USA, dilution ratio of WB,1:1000, IHC,1:200), Bak (12150S, CST, USA, dilution ratio of WB,1:1000, IHC, 1:200), Caspase-3 (9662S, CST, USA, dilution ratio of WB,1:1000, IHC, 1:1000), Bcl-2 (15071S, CST, USA, dilution ratio of WB,1:1000, IHC, 1:500), Lats1 (ab243656, Abcam, USA, dilution ratio of WB,1:1000), Mst1 (ab317410, Abcam, UK,dilution ratio of WB,1:1000), Sav1 (ab307698, Abcam, UK,dilution ratio of WB,1:1000), p -YAP (ab313464, Abcam, USA, dilution ratio of WB,1:500), Purification:Article Title: Topoisomerase I Inhibition in ETV4-overexpressed Non-Small Cell Lung Cancer Promotes Replication and Transcription Mediated R-Loop Accumulation and DNA Damage. Article Snippet: .. In addition, Article Title: Topoisomerase I Inhibition in ETV4‐overexpressed Non‐Small Cell Lung Cancer Promotes Replication and Transcription Mediated R‐Loop Accumulation and DNA Damage Article Snippet: .. In addition, Western Blot:Article Title: Sestrin2 improves intestinal ischemia-reperfusion injury through the TFEB-mediated autophagy/lysosomal pathway. Article Snippet: Sestrin2 is a stress-inducible protein that exhibits protective effects against ischemiareperfusion injury in various organs.. However, the specific roles and mechanisms of Sestrin2 in intestinal ischemia-reperfusion (IIR) injury have yet to be fully elucidated.. The present study aims to investigate the role of Sestrin2 in intestinal IIR injury and its underlying mechanisms. |
![( A ) Metabolism pathway enrichment analysis of DEGs in DAM2 microglial cluster. ( B ) Lipid/cholesterol metabolism–related DEGs in DAM2. mTOR, mechanistic target of rapamycin. ( C ) Retinal section stained with Filipin (free cholesterol; magenta), Iba-1 (microglia; green), and <t>Histone</t> <t>H3</t> (nuclei; white) antibodies and quantification of Filipin + microglia in the ONL of the retinas from rd10 retinas administrated with AAV- Cx3cr1-Tpm1 -EGFP or control vector ( n = 5 mice per group). Yellow arrowheads indicate Filipin + microglia. Scale bar, 20 μm. For image acquiring, three views in each retinal section at 100 μm (central), 1 mm (middle), and 1.8 mm (peripheral) from the optic nerve head along the dorsal and ventral directions were captured. ( D ) qPCR validation of Tpm1 and lipid metabolism regulators ( Apoe , Fabp5 , Apoc1 , Plin2 , Lypla1 , Sp1 , and Soat1 ) in primary microglia transfected with Tpm1 plasmid versus control ( n = 3 independent experiments). ( E to G ) Fluorescent colocalization analysis of lipid and cholesterol accumulation (Nile Red: lipid droplets; BODIPY C11: neutral lipids; Filipin: free cholesterol) in microglia following Tpm1 overexpression and Apoe/Fabp5 knockdown. Cell percentages of Nile Red + , BODIPY C11 + , or Filipin + microglia were quantified ( n = 3 independent experiments). Scale bars, 20 μm. ( H ) Transcriptional profiling of inflammatory mediators in dual-modified microglia ( n = 3 independent experiments). ( I ) Schematic model: Tpm1 up-regulation drives Apoe/Fabp5 -dependent lipid accumulation in microglia, promoting neuroinflammatory responses and photoreceptor degeneration in RP. Data represent means ± SEMs. Statistical significance determined using unpaired two-tailed Student’s t test (C) or one-way ANOVA with Tukey’s multiple-comparison test [(D) to (H)] (* P < 0.05, ** P < 0.01, *** P < 0.001, and **** P < 0.0001).](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_5182/pmc13215182/pmc13215182__sciadv.aea6467-f7.jpg)